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discreet

ALTAMOFEN

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$34,00

Characteristics

ACTIVE HALF-LIFE
5-7 days
Classification
SERM
Dosage
10-30 mg/day
ACNE
No
WATER RETENTION
No
HBR
No
HEPATOXITY
No
AROMATIZATION
No
Active Substance
Form
50 pills x 20 mg
Manufacturer

Description

 

  • Chemical name:(Z)-2-[4-(1,2-Diphenylbut-1-enyl)phenoxy]-N,N-dimethylethanamine
  • Formula: C26H29NO
  • Anabolic activity index: not a steroid
  • Androgenic activity index: not a steroid

 

 

ALTAMOFEN 20mg - ALPHA PHARMA

 

Alpha-Pharma Altamofen (Tamoxifen Citrate) belongs to a category and class of drugs known as selective Estrogen receptor modulators (SERMs). Selective Estrogen receptor modulators belong to an even broader class of drugs known as anti-estrogens. The other subcategory of drug under the anti-estrogens category is known as aromatase inhibitors (AIs), such as Aromasin (Exemestane) and Arimidex (Anastrozole). AIs and SERMs make up anti-estrogens. Aromatase inhibitors differ greatly from SERMs in their action and how they deal with the issues of estrogen control. The misunderstanding that SERMs, such as Altamofen and Clomid, serve to lower estrogen levels must first be addressed before delving into any further details.

This is a persistent rumor among the anabolic steroid using community that has begun to erode as of late, but the rumor still persists. SERMs serve to block the action of Estrogen at the receptor sites in breast tissue by occupying the receptor sites in place of Estrogen so that Estrogen itself cannot exert its effects there through receptor site binding. Conversely, SERMs will also act as Estrogens at receptor sites at other cells in other areas of the body (the liver, for example in Altamofen’s case). SERMs do not lower circulating levels of Estrogen in blood plasma. Aromatase inhibitors serve to do this by eliminating the production of Estrogen through binding to and disabling the aromatase enzyme, which is the enzyme responsible for the conversion (or aromatization) of androgens into Estrogen.

Altamofen is specifically a non-steroidal SERM belonging to the triphenylethylene family of compounds that exhibits both Estrogen agonist and Estrogen antagonistic effects on the body. This means that although Altamofen might block the effect of Estrogen at the cellular level in certain tissues, it can enhance Estrogenic effects in other areas of the body. These can be positive effects as well as negative effects. Altamofen exhibits Estrogenic effects in the liver, for example, which for all intents and purposes is a positive effect, as its effects here result in a positive change in cholesterol profiles (something desired by many). The area of concern with Altamofen is particularly in breast tissue, where it serves to act as an anti-Estrogen in this area (and a very strong one at that).

This is where it is utilized in medicine, as a first-line treatment in Estrogen-responsive female breast cancer patients. It is even utilized in females that do not possess breast cancer, but are known as being in a high risk category (due to hereditary genetics or otherwise) as a preventative measure. It is through its medical application that one can easily see how such a compound would easily attract the anabolic steroid using bodybuilding and athletic community, as one of the more prominent concerns among anabolic steroid users is that of the Estrogenic effects caused by the use of aromatizable androgens (such as Testosterone, Dianabol, Boldenone, etc.), mores specifically: gynecomastia (the development of breast tissue). If gynecomastia is left unchecked, it can develop and grow beyond a reversible point whereby the only option for removal is that of surgery.

Altamofen (Tamoxifen Citrate, aka known as Noldvadex or simply Nolva) possesses other very desirable effect, such as its ability to increase circulating levels of endogenous gonadotropins Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH), and through this, increasing the endogenous production of Testosterone. This is a perfect example of the mixed Estrogen antagonistic and agonistic properties of Altamofen, where it works to block the ability for Estrogen to bind to receptors at the hypothalamus. This results in the manipulation of the negative feedback loop of the HPTA (Hypothalamic Pituitary Testicular Axis), and results in the increased output of gonadotropins (the hormones that signal the testes to begin or increase the output of Testosterone production). It is for this reason that Altamofen is considered an absolutely essential component of a PCT (Post Cycle Therapy) program during the weeks following anabolic steroid use in order to restore the function of endogenous Testosterone production. Altamofen’s effect in these aspects will be further covered in detail in this profile.

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